Cholesterol-Induced Phenotypic Modulation of Smooth Muscle Cells to Macrophage/Fibroblast–like Cells Is Driven by an Unfolded Protein Response | Arteriosclerosis, Thrombosis, and Vascular Biology
Graphical Abstract
NAADP/SERCA3-Dependent Ca2+ Stores Pathway Specifically Controls Early Autocrine ADP Secretion Potentiating Platelet Activation | Circulation Research
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text
Activation of p42/p44 Mitogen-Activated Protein Kinase and Contraction by Prostaglandin F2α, Ionomycin, and Thapsigargin in Cat Iris Sphincter Smooth Muscle: Inhibition by PD98059, KN-93, and Isoproterenol | Journal of Pharmacology and Experimental
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug
IJMS | Free Full-Text | Dual role of Endoplasmic Reticulum Stress-Mediated Unfolded Protein Response Signaling Pathway in Carcinogenesis
Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the End—of Thapsigargin Prodrug-Based Cancer Therapeutics
Cancer metabolism and mitochondria: Finding novel mechanisms to fight tumours - eBioMedicine
ER-phagy: mechanisms, regulation, and diseases connected to the lysosomal clearance of the endoplasmic reticulum | Physiological Reviews
Ca2+ release or Ca2+ entry, that is the question: what governs Ca2+ oscillations in pancreatic β cells? | American Journal of Physiology-Endocrinology and Metabolism
Promising therapeutic effect of thapsigargin nanoparticles on chronic kidney disease through the activation of Nrf2 and FoxO1 - Figure f8 | Aging
From Plant to Patient: Thapsigargin, a Tool for Understanding Natural Product Chemistry, Total Syntheses, Biosynthesis, Taxonomy, ATPases, Cell Death, and Drug Development | SpringerLink
MANF Ablation Causes Prolonged Activation of the UPR without Neurodegeneration in the Mouse Midbrain Dopamine System | eNeuro
Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
Calcium Influx Caused by ER Stress Inducers Enhances Oncolytic Adenovirus Enadenotucirev Replication and Killing through PKCα Activation: Molecular Therapy - Oncolytics
Coupling endoplasmic reticulum stress to the cell death program | Cell Death & Differentiation
Thapsigargin - Wikipedia
Thapsigargin induces apoptosis when autophagy is inhibited in HepG2 cells and both processes are regulated by ROS-dependent pathway - ScienceDirect
Peptide–drug conjugate-based novel molecular drug delivery system in cancer: Trends in Pharmacological Sciences
Frontiers | The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and Neurodegeneration in Parkinson's Disease
